CATDOLL : CATDOLL: What causes Ankara disease in chickens?

1. What causes Ankara disease in chickens?

1. Pathogen: The disease was confirmed by laboratory tests to be adenovirus infection. As for adenovirus, what we are most familiar with is inclusion body hepatitis, which has existed in my country for at least 30 years, and its onset has been known by most veterinary workers. The virus that caused the disease this time is homologous to inclusion body hepatitis, both of which are adenovirus infections, but the serotype has changed. According to relevant experts, it may be serotype 4. In the past, we found that inclusion body hepatitis generally had more other types. Therefore, this outbreak also belongs to inclusion body hepatitis, but the serotype is different. 2. Epidemiology: The disease mainly occurs in broilers, 817, and hens aged 1 to 3 weeks, and can also be seen in broiler breeders and laying hens, among which chickens aged 5 to 7 weeks are the most common. Because it was first discovered in Karachi, Pakistan, near Ankara, it is also called Ankara disease. Most of the chickens that were infected began to die at the age of 3 weeks, reaching a peak at the age of 4 to 5 weeks, and the peak lasted for 4 to 8 days, and the death rate decreased at the age of 5 to 6 weeks. The course of the disease is 8 to 15 days, and the mortality rate is 20 to 80%, generally around 30%. The disease can be transmitted vertically or horizontally. After infection, chickens can become lifelong carriers of the virus and can excrete toxins intermittently. As for this disease, it is now mainly concentrated in Shandong, Henan, Jiangsu and other places. 3. Clinical symptoms: The liver, spleen and kidneys are mainly affected. The incubation period is short and the onset is fast. It is characterized by sudden collapse without obvious signs, depression, feathers in bundles, respiratory symptoms, shaking nose, rapid breathing, some with rales, yellow loose feces, neurological symptoms, legs scratching, and death within minutes. 4. Anatomical lesions: The myocardium of sick chickens is soft, and the pericardium accumulates light yellow transparent exudate. The liver is swollen, congested, with blunt edges, brittle texture, yellow color, and necrosis. The kidneys are enlarged, pale or dark yellow. The lungs are congested and edematous, with a black appearance, some of them have air sac inflammation (suspected of other infections), the spleen is slightly enlarged, the muscles are pale, the visible mucosa becomes lighter, and the intestinal changes are not obvious. 5. Diagnosis: A preliminary diagnosis can be made based on the symptoms and pathological changes. The diagnosis can be made by seeing basophilic nuclear inclusions in the liver section. It is confirmed that chicken embryo liver cell culture is required for virus isolation. 6. Prevention and control measures: The main treatment plan for this disease is symptomatic treatment, and the main treatment ideas are a. Antiviral. B. Protect the liver and kidneys, c. Strengthen the heart and diuresis, d. Control secondary infection. In addition, the main prevention and control measures in the epidemic area in terms of feeding and management are still to prevent the disease through vaccination in order to effectively control and purify the disease. Prevention should also start with breeder chickens. This disease has a strong vertical transmission. In case of emergency, emergency vaccination with self-organized inactivated vaccines can effectively reduce the mortality rate. Prevention and control of this disease also requires reducing stress, preventing heat stress, reducing density, increasing ventilation, ensuring insufficient oxygen required, and reducing negative pressure to prevent insufficient oxygen, leading to cardiopulmonary failure. It also absorbs mold and toxins to improve the body's immunity. You can use Huizhongde's Yuhuang oral liquid for treatment

2. How to treat Chicken Ankara?

1. Protect the liver and kidneys and act as a diuretic. Since this disease can cause inflammatory changes such as hepatomegaly and kidney enlargement, pericardial effusion, etc., glucose and Wuling San are used!

2. Strengthen management, reduce density, enhance ventilation, clean the chicken coop in time and rotate and disinfect frequently!

3. After many experiments, it has been confirmed that Longda tripeptide injection combined with polypeptide factor drinking water has a good therapeutic effect on Ankara virus!

It depends on the specific situation!

3. What is Ankara Falls?

The Kalandula Falls in Malanje Province, Angola, is the second largest waterfall in Africa. It exudes unparalleled wild charm and is one of the most famous natural landscapes in Angola. It is located on the Lukala River in Malanje Province. It is 344 feet high and 1,300 feet wide. It is on the tributary of the Guangza River, about 420 kilometers away from the capital Luanda. If you have the opportunity to go to Africa, it is worth a trip here.

4. Which country is Ankara in?

Türkiye

Ankara is the capital and second largest city of Turkey. Ankara is an ancient city with a long history. There are many historical sites in the city, such as the Julian Column and the Temple of Augustus from the Roman period; the castle and cemetery from the Byzantine period; the Aladdin Mosque from the Seljuk period; and the Mehmed Pasha Market and the Mehmed Market from the Ottoman period.

5. How long can the Chicken^Ankara virus survive in the soil?

The survival time of Chicken Ankara virus in soil depends on many factors, and different research results are not consistent. Generally speaking, the survival time of Chicken Ankara virus in soil is related to factors such as soil pH and humidity. Under normal circumstances, the virus survives in soil for weeks to months. It is reported that scientists are conducting more research to gain a deeper understanding of the life characteristics of Chicken Ankara virus and its performance in the environment.

6. What is chicken aspergillosis?

Chicken aspergillosis is mainly caused by Aspergillus fumigatus, Aspergillus flavus and Aspergillus niger. Under suitable temperature and humidity conditions, the fungus adheres to feed, bedding and feces and reproduces. It mainly harms chicks. Chicks will be infected when they eat feed contaminated with mold spores and live on bedding. It will present an acute outbreak type, and the mortality rate can be as high as about 30%. Adult chickens are less infected. In addition, during the incubation process, Aspergillus can enter the egg through cracked or washed eggshells, causing embryonic death.

Chicks are mostly infected within 5 days of age. Chicks older than 5 days have enhanced resistance to Aspergillus and the infection rate is greatly reduced. The main symptoms of chicks infected with Aspergillus are difficulty breathing, stretching the neck and raising the head, rapid breathing or gasping with the mouth open, and a large amount of mucus secretions in the mouth. The sick chicks have anorexia, lack of energy, lethargy, and like to drink water. In the later stage, diarrhea occurs, and they quickly become emaciated and die of exhaustion.

Autopsy shows that the air sacs and lungs of the sick chicks have gray or yellowish nodules ranging in size from the tip of a needle to a grain of rice, and liver-like changes are found in some parts of the lungs. Adult chickens infected with Aspergillus often have a chronic course, and the symptoms are basically similar to those of chicks. Commonly, sick chickens have pale combs, bloody and thin feces, become emaciated, and have decreased egg production. Arthritis often occurs in later chickens.

7. What is the cause of chicken biting chicken?

Pecking

Also known as pica, it is a very complex taste abnormality syndrome caused by multiple nutrient deficiencies or other metabolic disorders. Some inappropriate environmental and management factors are also important causes of pica. Pica can occur in chickens of all ages and breeds, but it is more likely to occur in neurotic laying hens. Once a chicken flock develops pica, even if the triggering factors disappear, the bad habit will often continue, causing injury, disability, and death to the chickens, often causing great economic losses to the chicken farmers.

8. Are you specialized in chicken diseases?

I think chicken disease is professional. Because chicken disease is a professional field. So it is professional

9. What diseases are chickens prone to?

1. Bacterial diarrhea The weather is cold and hot, resulting in poor intestinal conditioning, a large number of pathogenic microorganisms proliferate, and diarrhea. The chickens are in good spirits, but grow slowly, with pale combs. Some chickens have white strips or mucous feces, which contain undigested feed and are slightly yellow in color. Some chickens are depressed, shrink their heads, tremble, run around, scream easily, and then become paralyzed. The incidence rate of chickens is about 1%. Prevention and control measures: (1) Isolate sick chickens, eliminate residual chickens, remove litter, and completely disinfect. (2) Pay attention to both heat preservation and ventilation. (3) Suggest a medication plan and use microecological preparations to restore intestinal function. Strengthen the feeding and management of the chickens to keep the chickens strong and do a good job of epidemic prevention and disinfection. (4) Drug prevention: Commonly used drugs include penicillin, chlortetracycline, norfloxacin, tetracycline, oxytetracycline, streptomycin, etc., which have effective effects. The drugs can be mixed with water, mixed with feed, and administered to each chicken. For sick chickens that refuse to eat or drink, injection should be used (50,000 to 100,000 units of penicillin per chicken, twice a day, for 2 to 3 consecutive days). (5) Immunization: If the disease is not prevalent in the chicken farm, vaccination with bacteria is generally not necessary. In prevalent areas, vaccination with bacteria has a certain effect.

2. Fowl pox is caused by fowl pox virus. When fowl pox occurs in laying hens, the egg production rate decreases. The chickens' appetite, spirit and feces are basically normal. Skin-type fowl pox has characteristic papule-like nodules on the comb and wattles, which generally do not cause death; mucosal-type fowl pox has yellow-white cheesy pseudomembranes attached to the mouth and throat, and there is a "crackling" sound when breathing. During autopsy, local pox can be seen in skin-type fowl pox; in addition to the yellow-white pseudomembranes in the mouth and throat, mucosal-type fowl pox can also be seen in the tracheal mucosa. There are no lesions in other internal organs. Prevention and control measures: (1) The most reliable way to prevent fowl pox is to vaccinate with fowl pox vaccine. (2) Keep the chicken house clean, dry, ventilated and light-permeable, and disinfect it regularly. In particular, mosquito prevention and control should be done well. (3) Antibiotics are used to treat fowl pox to prevent secondary infection. The scabs on the skin are generally not treated.

3. Chicken leukocytosis is a disease caused by leukocytosis invading the tissue cells of blood and internal organs. Sick chickens are depressed, stand with eyes closed, have diarrhea, and have a reduced or no appetite; their feces are green and often bloody; they are anemic, emaciated, have pale crowns, and grow slowly; most sick chickens suddenly cough up blood and have difficulty breathing before death; the egg production rate of laying hens drops sharply. During autopsy, it can be seen that the chicken's crown and wattles are pale, the meat color becomes lighter, and there is extensive subcutaneous bleeding all over the body; the liver is enlarged and earthy yellow, with yellow-white nodules as large as millet grains to soybeans; the kidneys are swollen and bleeding, and there are a lot of blood clots on their surface; the spleen has bleeding spots; the pancreas, gizzard, and abdominal fat of some chickens have bleeding spots; the lungs are bleeding, and there are blood clots in the trachea and bronchi.

4. Infectious rhinitis in chickens Infectious rhinitis in chickens is characterized by inflammation of the nasal cavity and sinuses, sneezing and facial swelling. The more obvious symptoms are facial swelling, serous and mucous secretions in the nasal cavity and sinuses, conjunctivitis, swelling of the tissue around one eye socket, severe blindness, obvious edema of the wattles, and difficulty breathing when the inflammation of the upper respiratory tract spreads to the trachea and lungs. Prevention and control measures: (1) Vaccination. (2) Feeding management: Strengthen feeding management, improve ventilation conditions in chicken houses, reduce ammonia content in the environment, implement a full-in and full-out feeding system, completely disinfect the house after emptying it, and wait for a period of time before introducing new chickens, and do a good job of veterinary hygiene and disinfection inside and outside the chicken house. (3) Choose sulfonamide drugs to treat this disease. Chinese herbal medicine: 100 grams each of Angelica dahurica, Saposhnikovia divaricata, Leonurus japonicus, Prunus mume, Polyporus umbellatus, Terminalia chebula, and Alisma orientalis, 80 grams each of Cyperus rotundus, Platycodon grandiflorum, Scutellaria baicalensis, Pinellia ternata, Ginger, Leonurus japonicus, and Licorice. Crush and sieve, mix well, and it is the dosage for 100 chickens for 3 days, that is, an average of 42 grams per chicken per day. Mix with feed and feed for 9 consecutive days.

10. How to identify chicken diseases by looking at chicken manure?

Normal chicken manure is in strips, with a layer of white urate on the surface, and moderate hardness. Its color may vary depending on the type of feed, mostly gray-green or sauce-yellow. If it is too hard or too thin, it is caused by insufficient or excessive drinking water. But if it is too soft, it is because there is too much bran and wheat in the feed. If the chicken manure is abnormal in quality, quantity, shape and color, it may be caused by disease.

There are several types of abnormal chicken feces: Flesh-red feces: The shape is like rotten meat, which is formed by the shedding of intestinal mucosa. It is more common in chickens suffering from coccidiosis, tapeworm disease, ascariasis and in the recovery period of enteritis. Bloody feces: The feces are black or dark brown, which is common in upper gastrointestinal bleeding; the feces are red or bright red, which is more common in lower gastrointestinal bleeding. Yellow sulfur feces: The surface of the feces is covered with a layer of yellow or light yellow urine.

That is because the liver lobule is damaged, which affects the excretion of bile, causing bilirubin to enter the blood and be excreted through urine. It is more common in appendicitis and hepatitis. Green sticky and foul-smelling feces: The feces are dark green, which is caused by the mixture of bile and intestinal shed tissue cells. It is more common in fowl cholera, Newcastle disease, laryngotracheitis, etc. Thin feces: The chicken digests normally, but the feces contain a lot of water and are not formed. It is mostly caused by a sudden increase in water intake during hot weather, too much salt in the feed, mild infection of E. coli, and slightly toxic substances in the feed.

Rusty watery stool: Rusty watery stool mixed with urate, and sometimes mixed with incompletely digested feed. This is caused by severe intestinal bleeding. It is often seen in diseases that cause gastrointestinal bleeding, such as early Newcastle disease poisoning. Milky stool: Milky white, watery, like milk poured on the ground, often seen in mucosal congestion and mild enteritis. White watery stool: Sticky, often sticky to the chicken's anus, often seen in pullorum.

White watery stool: The stool is watery and mixed with white urate particles. It is more common in chickens with no appetite, paralysis and uremia. This is caused by the absence of food in the digestive tract and the stool is urate.